דרוש מידע טרשת נפוצה

הנושא בפורום 'איש את רעהו' פורסם ע"י y t, ‏26/5/19.

  1. y t

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    להתמודדות עם קרוב משפחה החולה בטרשת נפוצה במצב מתקדם נשמח לקבל את המידע הבא:
    א. טיפולים אלטרנטיביים למניעת המשך התדרדרות...
    ב. פיזיוטרפיסט טוב שיכול לבוא לביקורי בית באיזור המרכז...
    ג. טיפול בחרדות כתוצאה מהמחלה, שמשפיעות בעיקר על התנועה [הליכה וכו']...
    בתודה מראש
     
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    מציעה ליצור קשר עם האגודה הישראלית לטרשת נפוצה (כך הם נקראים אם אינני טועה)
    יש להם הרבה מידע והרבה עזרה.

    שמעתי על שני טיפולים שעוזרים למניעת ההדרדרות, אבל הם לא אלטרנטיביים.

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    יש טיפול שעושים בסין שמחליפים את כל הדם של החולה בדם שלוקחים מחבל הטבור בלידות - נקרא דם טבורי.
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    האגודה כמו שכתבו יש להם מידע
    ויש עוד ארגון משו נראה לי לנשים אברר מחר

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  5. y t

    y t משתמש רשום

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    איזה טיפולים? אפשר יותר פרטים...
     
  6. y t

    y t משתמש רשום

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    ידוע לכם על אנשים שעברו את הטיפול וראו תוצאות?
     
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    https://mssociety.org.il/
     
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  8. אימא של

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    כן
     
  9. ד.נ.

    ד.נ. משתמש צעיר מוזיקה ונגינה

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    אני עברתי טיפולים של ״פלסמה פרזיס״ זה כמו דיאליזה,זה לא מחליש כמו דיאליזה .זה מחליף את ה״פלסמה שבדם, דהיינו החלבון שהוא נושא את הנוגדנים של המחלה .בהדסה יש מחלקה של אישפוז יום מצוינת !שם עשיתי את הטיפולים ,הבעיה שהקופה צריכה לאשר זאת.אני מציעה לכם לפנות לפר קרוסיס מנהל מחלקת נוירואימונולוגיה בהדסה,הוא למעשה מנהל של האגודה הישראלית לטרשת נפוצה ,הוא אדם נפלא!!וחושב מחוץ לקופסא והוא שלח אותי לטיפול הנ״ל,זה עזר לי מאוד! .הבעיה אצלו זה התור, דרך הזימון תורים בשר״פ תקבלו תור לפחות בעוד שנה! יש עסקן רפואי שהוא מיודד עם הרופא ויש לו דלת פתוחה אצלו ואפילו יותר ,הוא יכול לסדר תור מהר מאוד, כמובן שצריך להסביר לו את תכיפות הנושא,אם אתם מעונינים אפשר לכתוב כאן פרטי מייל או מספ טלפון ואני אתן לכם את המספר של העסקן הרפואי .רפואה שלמה
     
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  10. סימפטי

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    יש באירופה אני לא זוכר באיזה מדינה, שעושים איזה טיפול משהו כמו צינתור וזה עוזר לאחוז מסויים של חולים, הם טוענים שטרשת נפוצה זה בעצם חסימות בעורקים במקומות מסויימים.
     
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  11. סימפטי

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    Multiple Sclerosis & Myasthenia Gravis
    Dr. Klenner also turned his attention to other nervous system diseases. In a paper entitled, “Response of Peripheral and Central Nerve Pathology to Mega-doses of the Vitamin B complex and other Metabolites,” he focuses on Multiple Sclerosis and Myasthenia Gravis. (Journal of Applied Nutrition, Vol. 25, #304, 1973).

    He felt fatigue was the key to the understanding of the nervous system and its physiology. Substances are consumed for the production of energy in the muscles. Products of this process accumulate in the tissue. Some diseases will prevent this use of available energy. The junction between neuron and neuron and the connection between motor nerves and the fibers of skeletal muscle are the two locations for normal fatigue.

    Plants will wilt if fatigued; improper atmosphere and inadequate soil are responsible. Animals and humans need food, oxygen and faith to stay alive and healthy. He felt a sharecropper working in fresh open air would be less fatigued than a factory worker. Oxygen supply has much to do with fatigue.

    If a muscle is repeatedly stimulated, it will become so exhausted it will fail to respond. Either the glycogen is used up, or the lactic acid has accumulated to a poisonous level.

    (At this point he describes the aerobic and anaerobic metabolism of muscles. Phospho-creatine, adenosine triphosphate, calcium, magnesium and stored glycogen are all necessary for muscle function. Oxygen and small amounts of protein play a part in muscle contraction. Acetylcholine and its esterase are essential; too much or too little of any of these substances may prevent or slow down muscle action.)

    Myasthenia Gravis is a disease in which too much pyruvic acid, due to faulty metabolism, affects the interaction of acetylcholine at the junction of the nerve and the muscle. He felt at that time that Multiple Sclerosis was due to “sluggish and bizarre muscle activity due to the inability to utilize essential factors because of mechanical and chemical road blocks.”

    He felt chemical fatigue was common. Body lassitude is the result of ingestion of sedatives, hypnotics, tranquilizers and even sodium bicarbonate. The latter can displace oxygen from hemoglobin, cutting down oxygenation of tissues. But Vitamin C will prevent this type of energy loss. Smoking aggravates this fatigue.

    A person’s muscle exhaustion point is determined by his oxygen absorbing and carbon dioxide discharging ability. At rest we use 200 to 300 cc of oxygen per minute. With sudden exertion this will rise to 2000 to 4000 cc. The more oxygen absorbed, the more lactic acid will be removed. Efficient use of oxygen is the key to adequate energy production and removal of wastes.

    He described mental fatigue, active and passive. Passive is neurasthenia or brain fog: sensations of pressure in the head, poor memory, loss of ability to concentrate, irritability of temper, insomnia, anorexia and a variety of aches and pains.

    Active mental fatigue is caused by continuous work, and this change is due to the sensory-motor exhaustion and not the mental work per se. The primary area of fatigue is at the synapses which beg only diversion of interest and activity.

    Adequate oxygen is assured if the lungs and hemoglobin are normal, but also by taking 10 to 30 grams of ascorbic acid by mouth every 24 hours. Oxygen is released for tissue use when ascorbic acid becomes dehydroascorbic acid. Enzymes are necessary to make all these reactions possible. Genetic faults manifest themselves through enzymatic deficiencies.

    He outlines the nineteen stops from glucose to pyruvic acid which provides energy. This energy release depends upon oxygen and, Dr. Klenner emphasized, it is important to maintain good ventilation capacity, and, of course, a substantial intake of Vitamin C.

    He felt pyruvic acid metabolism was important for the understanding of Myasthenia Gravis. Coenzyme A (COA, the active form of pantothenic acid) is in limited supply in M.G. It, COA, intercepts pyruvic acid at the end point of glucose metabolism. Another enzyme, cocarboxylase, splits the carboxyl group (COOH) away from pyruvic acid to form CO2 and free hydrogen. The remaining two carbon fragment (acetate) join with coenzyme A to form acetyl coenzyme A. A high energy package named NADH2 is formed from the carboxyl group from pyruvic acid and a sulfur group from coenzyme A.

    Thiamin is important in all this energy production as two molecules of thiamin combined with two molecules of phosphoric acid become cocarboxylase. This enzyme must be present for the continuance of the metabolic cycle. When thiamin is deficient, pyruvates and lactate accumulate, and at the neuromuscular junction the nerve end plate becomes swollen and poorly operative. That same enzyme is necessary for the syntheses of acetylcholine, the neurotransmitter that initiates muscle contraction. “Thiamin deficiency inhibits lactic acid metabolism.” A thiamin deficiency means a cocarboxylase deficiency. Liver enzymes are mainly responsible for the phosphorylation of thiamin to cocarboxylase. Liver disease would obviously reduce this synthesis. “The activity of choline esterase (breaks down acetylcholine) is inhibited by this same double thiamin unit.” (See also p. 20.)

    In the conversion of fatty acids to energy some of the same enzymes are necessary: coenzyme A, hydrogen carriers (niacin-adenosine-dinucleotide) and Vitamin C. The latter acts as a hydrogen transport.

    He puts Myasthenia Gravis and Multiple Sclerosis in the same therapeutic group as he found thiamin was the key to the therapy. M.G. is a genetically transmitted disease and M.S. is triggered by a virus and mimics poliomyelitis. Nerve damage in M.S. is due to microscopic hemorrhages in the nervous system. During healing, scar tissue contracts clamping off capillary flow and nutrition. This wasting results in loss of the myelin sheath protection.

    He felt that remyelinating these damaged nerves was every bit as hopeful as the myelination that occurs normally in infancy with nothing more spectacular than breast milk. It requires two years of treatment to repair the damage caused by one year of the disease.

    He cites works in the late 1930s by Stern at Columbia University who used thiamin intraspinally for the treatment of Multiple Sclerosis with astonishing results. After 30 mg of thiamin was injected into the spinal canal of paralyzed MS. victims, they had a temporary remission. They could walk for a while. And Stern felt it was a B1 avitaminosis. It was known at that time that polyneuritis can cause degeneration of myelin sheaths.

    Dr. Klenner felt that both M.G. and M.S. were basically a disturbance of supply and demand and not a functional defect nor impaired diffusion. He followed the belief of Dr. Leon Rosenberg (Yale) who distinguishes between vitamin deficiency diseases and vitamin dependency diseases. Some diseases would require 1000 times the calculated minimal daily requirement. Another investigator [Moore] used high intravenous doses of nicotinic acid (B3) in the control of M.S.

    Dr. Klenner’s protocol for M.G. and M.S. in the 1950’s:

    1. Thiamin, (B1), orally: 300 to 500 mg 30 minutes before meals and at bedtime. Intramuscularly: 400 mg daily. Intravenously: 1000 mg (or 20 mg per kg body weight) two to three times a week. A 20 cc to 30 cc syringe with a one inch 22 gauge (or smaller) needle is used. The patient is to be supine and the pulse counted as the solution is injected. If the pulse rises, the solution is being injected too rapidly. Thiamin can be toxic but as soon as it is phosphorylated (in seconds) it becomes cocarboxylase, a necessary enzyme. Benadryl® intramuscularly stops any allergic reaction. Dr. Klenner reassures us that if injected slowly, no problem is encountered. The preservatives are more likely to cause reactions than the thiamin.
    2. Niacin or nicotinic acid, (B3), orally: 100 mg to 3000 mg thirty minutes before meals and at bedtime. The dose should be enough to produce a strong body flush. As it dilates the blood vessels—“even those that have been compressed by scar tissue”—a greater amount of the nutrients reach the muscle and nerve cells. Dr. Klenner felt it would be better to have a constant flush.
    3. Pyridoxine, (B6), orally: 100 to 200 mg before meals and at bedtime. Intramuscularly: 100 mg daily. Lack of B6 causes anemia and neurological lesions. Intravenously: 300 mg. It is necessary for the metabolism of fatty and amino acids.
    4. Cobalamin, (B12), intramuscularly: 1000 mcg three times a week. B12 is a factor in the synthesis of myelin. In the treatment of neurological diseases, B12 reduces the requirement of choline.
    5. Ascorbic acid, orally: 10 to 20 grams are to be taken daily in divided doses. Vitamin C will prevent a superimposed infection and aids in metabolism.
    6. Riboflavin, (B2), orally: 25 mg before meals and at bedtime. Intramuscularly: 40 to 80 mg daily. It is essential for metabolism of carbohydrates and in the regulatory function of the hormones involved in carbohydrate metabolism.
    7. d-alpha tocopherol acetate, (Vitamin E), orally: 800 Units before meals and at bedtime. A deficiency results in demyelinization and distortion of the spinal cord nerves.
    8. Crude Liver, daily injections. It contains factors still unknown but essential in metabolism. (Not manufactured now.)
    9. Adenosine-5-monophosphoric acid. By adding this, all the chemistry dealing with cell metabolism is enhanced. It is essential to muscle function and, thus, energy.
    10. Choline, orally: 700 to 1400 mg after each meal and at bedtime. It is in fat and nerve tissue. Acetylcholine plays an important role in humoral transmission of nerve impulses to effector organs like muscles.
    11. Lecithin, orally: 1200 mg of soybean lecithin after each meal. Lecithin contains choline. It plays an important part in the structure of cell membranes. It is the lipid used in nerve tissue.
    12. Magnesium, orally: 300 mg after each meal. Muscle activity requires magnesium. It also serves as an enzyme activator.
    13. Calcium gluconate, orally: ten-grain tablets. Two tablets after each meal and bedtime. Intravenously: one gram twice weekly. Helps muscle activity.
    14. Calcium pantothenate, orally: 500 mg after each meal and at bedtime. This is a coenzyme A. It participates in the acetylation of amines and metabolism of carbohydrates and fatty acids.
    15. Aminoacetic acid, (Glycine), orally: one heaping tablespoon of the powder in a glass of milk four times a day. It is concerned with the syntheses of glutathione which is involved with intracellular oxidation and reduction. It stimulates the combustion of other tissue constituents. It has an adaptability in the detoxification process.
    16. The hemoglobin should be kept to at least thirteen grams.
    17. The diet is to be high protein, including two to three eggs for breakfast.
    18. One Theragran-M capsule daily for trace minerals.
    19. Dantrium to relieve tremors. Sysmmetrol to relieve stiffness.
    20. Zinc gluconate, orally: 20 mg three times a day helps Myasthenia Gravis.
    This treatment works dramatically in M.G. An abbreviated schedule can be effective. One gram thiamin four times a day, niacin, enough to produce a flush four times a day, 200 mg calcium pantothenate four times a day, 100 mg pyridoxine four times a day, 10 grams of C in divided doses, glycine one tablet four times a day. This treatment is effective, but the full therapy will afford more dramatic response.

    Dr. Klenner felt that most cases (80%) of Multiple Sclerosis had their origin in an illness—probably a coxsackie virus—compatible with a summer “flu”. He mentioned other theories of the etiology of M.S., but was convinced that the scar tissue that forms around the nerves and produces the symptoms “is the end result of microscopic hemorrhages following virus invasion.”

    He believed that in M.G. the thymus gland was hyperplastic in many cases, and that muscle antibodies might account for others, but the importance of the excessive pyruvates at the neuromuscular junction has to be recognized as the basic cause of the hypotonia.

    Here followed a number of a case histories of neurological diseases. One case of M.S. was of a male confined to a wheel chair in the hospital for two years. After a month of the treatment listed above his physician realized the improvement and sent him home. In three years he was free from the disease and remained so as he continued in a modified treatment.

    One M.G. case was of a male receiving prostigmine to which he was becoming unresponsive; thiamin was given intramuscularly along with other B vitamins three times a day. He was off the prostigmine in a year. He lived a normal life for eighteen years. He died of an unrelated cerebral accident.

    A woman with polyneuritis began her illness with pain, burning and jerking of her legs accompanied by a high fever for ten days. Paralysis on left side plus weakness of the hands. She received oral and intramuscular injections. In several months intravenous vitamins were begun. In sixteen months she began to move her right leg. In five years from the beginning of the illness she began to get around with knee braces and a walker. In one more year she was able to move about without a back brace. Dr. Klenner felt if she had had 200 grams of ascorbic acid early, she would not have had the paralysis. She was also given 300 mg ribonucleic acid four times a week.

    Another woman developed weakness in her extremities and was diagnosed as M.S. superimposed by a viral encephalitis. She was sent home with a wheelchair and was expected to die. She fully recovered on Dr. Klenner’s protocol and continued to take her supplements.

    A male, aged 28, developed numbness and loss of muscle control from the waist down about two years before he came to Dr. Klenner’s treatment. He also had loss of bladder control. Dr. Klenner felt he had M.S. and put him on the above treatment. He was so much better in five weeks that he stopped treatment but the symptoms returned in three weeks, so he went back on the full treatment. Within a year he was back to full employment and able to follow his hobby as a crack pistol shooter.

    A white 57 year old female began to be fatigued seven years before coming to Dr. Klenner. She had normal function after a night’s sleep but had drooping eyelids and could not chew food after a few bites. Some doctors had called it psychosomatic. But it was quite obvious to Dr. Klenner that she had M.G. After 1000 mg of thiamin and 300 mg of pyridoxine administered intravenously in ten minute intervals, she was able to chew and make facial movements for the first time in three years. She has no symptoms as long as she continues the Klenner program.

    He was quite definite: “Any victim of Multiple Sclerosis who will dramatically flush with the use of nicotinic acid and has not yet progressed to the stage of myelin degeneration, as witnessed by sustained ankle clonus, can be cured with the adequate employment of thiamin, B complex proteins, lipids, carbohydrates and injectable crude liver.” “We had patients in wheel chairs who returned to normal activities after five to eight years of treatment.” He also noted that if M.S. patients had a course of ACTH or cortisone, it extended the recovery period.

    He noted the peripheral neuritis that is due to thiamin deficiency is common in chronic alcoholism.

    “The treatment of M.G. is that of any pathology dealing with the interruption of the normal physiology of nerve cells.” He had found that after successfully treating poliomyelitis victims with Vitamin C, he had to follow up with B vitamins for the nerve repair. He found the same results when treating damage to the spinal cord, whether trauma or viral infection. B1 restores the ability of the nervous system to handle pyruvic acid and dextrose properly. Cocarboxylase may be the “food required for nerve life.”

    Since M.G. does not suffer the loss of myelin sheaths in vital areas, it does not have to be treated as rigorously as M.S. But the chemistry is more complex because muscles are involved. 900 different enzymes have been identified, therefore vitamin therapy must be intense. Of course, good liver function is necessary for good results. Dr. Klenner stumbled on a liver test: a test tube is filled with a morning urine specimen. In 24 hours there is usually a gelatinous mass accumulation at the bottom; the more the amount, the more the stress to the liver. Choline will prevent this from appearing. These are phosphates.

    In an article, “Fatigue—Normal and Pathological”, [Southern Medicine and Surgery, Volume III, #9, Sept. 1949], he had already had success with the vitamin treatment of MS. and M.G. Dr. Klenner felt that fatigue is a warning signpost along the road of infectious disease. Heavy muscular exercise throws a great burden on the defensive mechanisms. The tissue of the adrenal cortex of rats is increased in weight after repeated periods of exercise.

    He pointed out the importance of oxygen in the etiology of fatigue. If the air that is inhaled has but 0.1 percent of carbon monoxide, half the hemoglobin will be bound to the CO and unavailable for carrying oxygen to the tissues.

    Poorly oxygenated blood can come from drugs, analgesics, and even sodium bicarbonate. A deficiency of B1 will reduce tissue (which breaks down acetylcholine needed at the nerve ending to activate the muscle). Shots of it are to be given daily from one to three weeks and then a 15 mg tablet orally every six hours.

    B1, 100 mg intramuscularly three times a day are given along with oral glycine. The other members of the B complex were added.

    “Avitaminotic nerve fibers have a hunger for this vitamin (B1), and it is easy to know when the optimum return of function is obtained. When the nerve structure has been repaired, the patient will become irritable, the appetite will be lost and he or she will experience a sensation of heaviness and stiffness of the muscles of the extremities. Sufficient Vitamin C is then given by mouth to maintain optimum therapeutics.”

    As to M.S. the diagnosis is determined by the “evidence of lesions affecting chiefly the white matter, scattered in time and space: palsy of one of the oculomotor nerves, nystagmus, slight ataxia of arms, absence of abdominal reflexes and other scattered neurological anomalies (such as poor bladder control and patchy sensory changes).

    Subtle forms of encephalitis might cause changes in the nervous system preventing a normal supply of Vitamin B1 from reaching distal parts of the nervous system. He noted the increased incidence of M.S. after the encephalitis epidemic of 1920-26 and in 1934. Also unrecognized cases of poliomyelitis may be an important factor in the cause of avitaminotic symptoms in the central nervous system. This could happen in these disease conditions even with sufficient B1 in the diet; the vitamin is not diffused properly. Initially it is the virus and when that dies down, it is scar tissue blocking the circulation. The capillaries must be opened and extra B1 must be supplied with the protocol cited above.

    In a letter to the editor of the Tri-State Medical Journal, Oct. 1954, he boldly stated that he was curing Myasthenia Gravis. He seemed more definite about the biochemistry: pyruvic acid, if allowed to accumulate, will produce a cloudy swelling of the distal portion of nerves, and that the primary biochemical fault in B1 deficiency is the failure of the organism to metabolize pyruvic acid. Also he realized that creatine (needed for normal muscle function) is formed by the body when choline and urea combine. Choline is in short supply in M.G. unless supplemented orally. He felt glycine should be supplemented in the diet because it yields urea. Protein is needed in the diet to sustain muscle wear and tear. Tyrosine is needed to help turn ingested protein into usable amino acids and Vitamin C is essential in this reaction.

    This leads us to paper he put together in 1980. It was not published: “Multiple Sclerosis Diagnosis and Treatment Suggestions.”

    He again stated the origin was due to a childhood virus of the coxsackie group mimicking red measles. The initial illness was a severe lung infection, or an encephalitis which subsided only to recur as M.S. twenty to thirty year later. 70% of cases have the onset of their M.S. symptoms from the age of 20-40 years.

    40% will have optic neuritis as the initial symptom, then optic atrophy may follow. Most will notice double vision early. Weakness, loss of reflexes, numbness in fingers, dizziness, loss of position sense, feeling heat over spine, rheumatoid arthritis may occur concurrently (shortage of B vitamins), intention tremor, poor bladder control, and spastic paraplegia.

    His treatment suggestion for M.S. at this time (1980) consisted of:

    1. Thiamin HCl (Vitamin B1) one gram (1000 mg) taken thirty minutes before meals and at bedtime.
    2. Nicotinic Acid (Niacin; Vitamin B3) 50 mg to 300 mg, depending on flushing of skin, thirty minutes before meals and bed time.
    3. Riboflavin (Vitamin B2) 250 mg after meals and bed time.
    4. Pyridoxine (Vitamin B6) 100 mg after meals and bed time.
    5. Calcium pantothenate (pantothenate acid/Vitamin B5) one gram after meals and bed time.
    6. Lecithin. 1200 mg (19 grains) one capsule after meals and at bed time with two percent milk.
    7. Vitamin A (palmitate) one 50,000 unit capsule after breakfast and supper.
    8. Vitamin E (d-alpha tocopheryl acetate) 400 I. units. Four capsules at bedtime.
    9. Niacinamide (Vitamin B3 amide) 500 mg. tablets. One after meals.
    10. Magnesium oxide 300 mg tablet. One tablet after meals and before bed time.
    11. Trinsicon or Feosol. One capsule twice daily or sufficient to maintain a hemoglobin of at least thirteen grams.
    12. Folic acid. Two milligrams after each meal. Only recommended when the hemoglobin will not respond to iron treatment.
    13. Sunflower seed oil capsules. One capsule after meals and bed time.
    14. Lipotriad. Three capsules yields 700 mg of choline. Two capsules after each meal. It is used as a methylating agent.
    15. Calcium gluconate, 10 grain tablets. Twelve tablets daily. May be omitted if patient can drink a quart of milk a day.
    16. Linseed oil capsules. One capsule after meals and at bedtime. Contains linolenic, oleic and linoleic acids.
    17. Muscle relaxants. Prescribed according to patient needs.
    18. Calcium Orotate (Vitamin B13) 500 mg tablet. One after meals and at bed time.
    19. Calcium pangamate, 50 mg tablet. One tablet twice daily.
    20. Protein supplement containing eighteen amino acids. One ounce in a glass of milk four times a day. Some of the above can be taken with this drink.
    [This list was originally numbered 1) to 22), with 11) and 12) missing –ed.]

    Intramuscular injection, given five to seven days each week.:

    1. 2 cc crude liver daily. (Hard to get now. I can’t find it.)
    2. 2cc Thiamin HCl, (B1), 400 mg daily.
    3. 1.5-2cc Pyridoxine, (B6), 150 mg daily. Add to B12.
    4. 1.5-2cc Cyariocobalamin, (B12), 1500 mcg daily. Add to B6.
    5. 1.5-2cc Riboflavin, (B2), 75 mg daily. Add to B3 amide.
    6. 1.5-2cc Niacinamide, (B3), 150 mg daily. Add to B2.
    Some of the above vitamins are given one to three times each week:

    Thiamin HCl, 1000 mg; Pyridoxine, 300 mg; Niacinamide, 500 mg; dilute these to 20 cc with saline solution or best, sodium ascorbate (250 mg/cc). Give slowly with a 23 gauge needle, one inch long. Pulse is taken during the injection; if the pulse rises, the injection speed is slowed.

    He found that RNA and DNA tablets, 100 mg of each, were helpful to some patients; one to three of each daily along with the other vitamins. Inositol, 500 mg, one to three times a day may help.

    Because of the large number of pills and capsules to be taken daily, Dr. Klenner suggested they be put into a blender along with a protein powder, milk, vanilla, and carob to make a tasty drink. They all might go down more easily.

    He cited some cases:

    1. Female developed weakness in extremities in 1961 (refer to page 48). She was sent home to deteriorate. Dr. Klenner began his program, and she is now cured and has been leading an active life for over 21 years. “The central nervous system can be regenerated, but it does require time. Ten years was given to the restitution of her entire nervous pathways.” She is “full of vim, vigor, and vitality.”
    2. Another woman had complete paralysis of both legs and left arm. She required a steel brace from hips to neck. After two years of this she was taken to Dr. Klenner and started on the above therapy. In sixteen months she could move her right leg and left arm. In three years she began to move her left foot and button her blouse. In nine years she could stand unaided. A modem day miracle, “Enzyme, co-enzyme, and metabolite theory is the correct approach to the rehabilitation of the central system.”
    3. In 1918 a male was diagnosed as M.S. because of blurred vision, numbness, and low back pain. In four months Dr. Klenner began his program and in six months the man was back driving the fire truck. He continued to improve and cut firewood during off hours. Early M.S. cases will respond quickly.
    4. Another female with dizziness, poor vision, lateral, and rotatory nystagmus (dancing eyeballs). The nausea was so profound; she could not swallow the oral vitamins. But after one year of the vitamin injections she could do the oral route. From not being able to read a billboard, she can now read large type books. The nystagmus is gone, but she needs a cane to ambulate.
    Complications
    Dr. Klenner reports on a few minor complications. Some diarrhea might have been due to sodium bisulfite. Induration after intramuscular injections was found to be due to the Vitamin C not being injected deeply enough into the muscle. (One had to be drained—a sterile abscess.) If the concentration was one gram to 5 cc it caused a vein spasm up the arm from the injection site in three cases. A thrombosis of the vein occurred in but one case. A minor face rash developed in a few that cleared after the C was stopped.

    Calcium seemed to enhance the effects of the C when both were give simultaneously. But a gram of just the calcium given intravenously can slow the heart rate to a dangerous degree.

    Safety
    He has some reassuring words for those who feel kidney stones are an automatic result of large doses of Vitamin C. He says in all cases a stasis of urine flow “and a concentrated urine appear to be the chief physiological factors.” Oxalic acid precipitates out of solution only from a neutral or alkaline solution—pH 7 to pH 10. Urine pH in those consuming ten grams of Vitamin C daily is about 6. Even in diabetics who take this large amount of C (10 grams), the urinary oxalate excretion remains relatively unchanged. “Vitamin C is an excellent diuretic. No urinary stasis; no urine concentration. The ascorbic acid/kidney stone story is a myth.” One more bon mot: “Methylene will dissolve calcium oxalate stones, if the patient is given 65 mg orally two to three times a day,” he learned from Medical World News (Smith, M.J.V., M.D.: Dec. 4, 1970).

    (90% of all stones are calcium stones. Calcium is soluble in acid media. Vitamin C acidifies the urine. Acid urine discourages the growth of bacteria. Although uric acid stones are theoretically possible with high doses of C and a low urinary pH, none have been reported.)

    A report in N.E.J.M. on 11 Feb, 1971 [Merton Lamden] suggested that large doses of C might cause diabetes in humans. The experiment was done in rats, but the dose translation in humans would have amounted to 5000 grams! [Paterson] Maybe there is a toxic dose. (Dr. Klenner at the time of that writing had been on 10 to 20 grams of C daily for eighteen years. No diabetes, and no kidney stones). This study has no relationship to the use of therapeutic doses of C.

    Lamden found that an ingestion of 9 grams of C/day resulted in oxalate spills of 68 mg. in the urine per 24 hours. Controls without C spilled 64 mg./24 hours. Not a big difference.

    He reiterates the safety of large doses of C. He states that plasma doses of greater than twenty times normal produce no ill effects. Diarrhea is the most common side effect of large doses. Some notice thickening of subcutaneous tissue is the C is not injected deeply enough into, the muscle. (That induration will eventually resolve.) Some will complain of venous irritation and spasm if the intravenous Vitamin C is too concentrated or too rapidly injected. (C mixed with calcium will reduce this irritation.) A rare thrombosis may occur if the concentration of the C is greater than 500 mg per cc. Some will faint if the injection is given too rapidly. (It is best to have the patient lie flat.) Large doses by mouth may cause a genital or anal rash and itch.

    He also showed how safe large doses of C were. He gave 200 patients 500 to 1000 mg of C every four to six hours for five to ten days. No laboratory abnormalities were found in blood or urine and no symptoms were noted except one percent who developed vomiting; he assumed from a hypersensitive stomach. And these patients had no virus infection to “assist in destroying the vitamin.”

    One volunteer received 100,000 mg in a twelve day period; no problems.

    Reluctance by Orthodox Medicine to Accept
    Dr. Klenner knew all this way back thirty to forty years ago. Why has the medical community taken so long to use this cheap, safe, and valuable tool to control infections? Dr. Irwin Stone, Dr. Linus Pauling, and Dr. Robert Cathcart have tried to popularize this method and were only met with poor press and ridicule. Are the drug manufacturers organized into a conspiracy too powerful to overcome? M.D. types will believe what is published in their favorite medical journals, but Vitamin C therapy studies are not seen in medical journals because much of the income to the publishers comes from drug manufacturers. Vitamin C use represents a threat to their income; it cannot be patented. Maybe if patients demanded the therapeutic use of Vitamin C from their doctors, the doctors would become familiar with its use and add it to their therapeutic tools. Their colleagues would hoot: “Ha ha, you are a quack. You were suckered into that.”

    The doctor could respond: “I didn’t want to, but the patient made me do it.”

    But the evidence for its use seems to be there, right in the medical literature, but how many read the Journal of Preventative Medicine?

    Dr. Klenner writes clearly and cogently. He is cheerful, even enthusiastic. And I find no bitterness due to the frustrations about the poor acceptance of his research by the medical establishment. He had done his own literature search and finds plenty of confirmation for his therapies in animal and human experiments.

    “Many physicians refuse to employ Vitamin C in the amounts suggested, simply because it is counter to their fixed ideas of what is reasonable.” The new products advertised by an alert drug company are okay to them. Dr. Klenner tells of many letters from doctors who used this C treatment on poliomyelitis—in patients, their own children and even themselves. They were cured.

    Dr. Klenner commented that if these spectacular results had been produced at a teaching and research center and then published, the medical community might pay some attention and the use of C would become standard and routine. “There is no doubt that physicians are being brainwashed with the current journal advertising.” He uses an appropriate quote from Herber Spencer, “… to keep a man in everlasting ignorance… condemnation without investigation.”

    He blamed the National Research Council who planted the concept in doctors’ brains that any dose above 125 mg per day is spilled by way of the kidneys. It was like any drug, the council implied, and more was no more effective than the dinky dose that protected the human from scurvy. Doctors do not seem to realize that the need for C is different “in each one of us either because of the individual kidney threshold level or because of greater requirements necessitated by pathology.”

    A Few Quotes
    He reminds us of Hippocrates. He felt that of several remedies physicians would choose the least sensational. Vitamin C meets those requirements.

    “Adults taking at least ten grams of ascorbic acid daily and children under ten at least one gram for each year of life will find that the brain will be clearer, the mind more active, the body less wearied, and the memory more retentive.”

    Another summary by Dr. Klenner: “I have never seen a patient that Vitamin C would not benefit.”

    He discovered the tremendous therapeutic power of Vitamin C to aid the immune system, to act as an antihistamine, and to neutralize toxins. Again, let us not forget what comes through after examining all these published reports: “Vitamin C should be given to the patient while the doctors ponder the diagnosis.”